Prolonged Ketamine Effects in Sp4 Hypomorphic Mice: Mimicking Phenotypes of Schizophrenia
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Terrence J. Sejnowski | Xin Wang | Xianjin Zhou | T. Sejnowski | Jared W. Young | M. Geyer | A. Pinto-Duarte | M. Behrens | Mark A. Geyer | S. Caldwell | Baohu Ji | Baohu Ji | Antonio Pinto-Duarte | Minjung Kim | Sorana Caldwell | Margarita M. Behrens | Xin Wang | Xianjin Zhou | J. Young | Minjung Kim
[1] M. Alegre,et al. Ketamine-Induced Oscillations in the Motor Circuit of the Rat Basal Ganglia , 2011, PloS one.
[2] R. Conley,et al. Ionotropic glutamate receptors and expression of N-methyl-D-aspartate receptor subunits in subregions of human hippocampus: effects of schizophrenia. , 2000, The American journal of psychiatry.
[3] A. Sampson,et al. Cortical deficits of glutamic acid decarboxylase 67 expression in schizophrenia: clinical, protein, and cell type-specific features. , 2011, The American journal of psychiatry.
[4] M. Goodell,et al. Genome-wide association study of recurrent early-onset major depressive disorder , 2010, Molecular Psychiatry.
[5] J. Kelsoe,et al. Impaired postnatal development of hippocampal dentate gyrus in Sp4 null mutant mice , 2007, Genes, brain, and behavior.
[6] M. Geyer,et al. Four factors underlying mouse behavior in an open field , 2012, Behavioural Brain Research.
[7] J. Kelsoe,et al. Transcription Factor SP4 Is a Susceptibility Gene for Bipolar Disorder , 2009, PloS one.
[8] D. Clair,et al. Association within a family of a balanced autosomal translocation with major mental illness , 1990, The Lancet.
[9] A. Sampson,et al. Gene Expression Deficits in a Subclass of GABA Neurons in the Prefrontal Cortex of Subjects with Schizophrenia , 2003, The Journal of Neuroscience.
[10] K. Nakazawa,et al. Postnatal NMDA receptor ablation in corticolimbic interneurons confers schizophrenia-like phenotypes , 2010, Nature Neuroscience.
[11] A. Sampson,et al. Decreased glutamic acid decarboxylase67 messenger RNA expression in a subset of prefrontal cortical gamma-aminobutyric acid neurons in subjects with schizophrenia. , 2000, Archives of general psychiatry.
[12] D. Javitt,et al. Recent advances in the phencyclidine model of schizophrenia. , 1991, The American journal of psychiatry.
[13] D. Supp,et al. Sp4, a member of the Sp1-family of zinc finger transcription factors, is required for normal murine growth, viability, and male fertility. , 1996, Developmental biology.
[14] Martin S. Taylor,et al. Disruption of two novel genes by a translocation co-segregating with schizophrenia. , 2000, Human molecular genetics.
[15] M A Geyer,et al. Disruption of prepulse inhibition and increases in locomotor activity by competitive N-methyl-D-aspartate receptor antagonists in rats. , 1999, The Journal of pharmacology and experimental therapeutics.
[16] H. Holcomb,et al. Effects of Noncompetitive NMDA Receptor Blockade on Anterior Cingulate Cerebral Blood Flow in Volunteers with Schizophrenia , 2005, Neuropsychopharmacology.
[17] Paul J Carlson,et al. A randomized trial of an N-methyl-D-aspartate antagonist in treatment-resistant major depression. , 2006, Archives of general psychiatry.
[18] Richard Redon,et al. Confirmed rare copy number variants implicate novel genes in schizophrenia. , 2010, Biochemical Society transactions.
[19] S. Kapur,et al. NMDA receptor antagonists ketamine and PCP have direct effects on the dopamine D2 and serotonin 5-HT2 receptors—implications for models of schizophrenia , 2002, Molecular Psychiatry.
[20] W. Singer,et al. Progress in Biophysics and Molecular Biology , 1965 .
[21] B. Moghaddam,et al. NMDA Receptor Hypofunction Produces Opposite Effects on Prefrontal Cortex Interneurons and Pyramidal Neurons , 2007, The Journal of Neuroscience.
[22] David A Lewis,et al. Glutamate Receptor Subtypes Mediating Synaptic Activation of Prefrontal Cortex Neurons: Relevance for Schizophrenia , 2011, The Journal of Neuroscience.
[23] S. Nakanishi,et al. Molecular cloning and characterization of the rat NMDA receptor , 1991, Nature.
[24] Carol A. Tamminga,et al. Subanesthetic Doses of Ketamine Stimulate Psychosis in Schizophrenia , 1995, Neuropsychopharmacology.
[25] B. Moghaddam,et al. Corticolimbic Dopamine Neurotransmission Is Temporally Dissociated from the Cognitive and Locomotor Effects of Phencyclidine , 1998, The Journal of Neuroscience.
[26] G. Rosenbaum,et al. Study of a new schizophrenomimetic drug; sernyl. , 1959, A.M.A. archives of neurology and psychiatry.
[27] P. Schofield,et al. Molecular evidence of N-methyl-D-aspartate receptor hypofunction in schizophrenia , 2012, Molecular Psychiatry.
[28] M. Geyer,et al. Reduced expression of the Sp4 gene in mice causes deficits in sensorimotor gating and memory associated with hippocampal vacuolization , 2005, Molecular Psychiatry.
[29] J. Krystal,et al. Subanesthetic effects of the noncompetitive NMDA antagonist, ketamine, in humans. Psychotomimetic, perceptual, cognitive, and neuroendocrine responses. , 1994, Archives of general psychiatry.
[30] Bita Moghaddam,et al. Activation of Glutamatergic Neurotransmission by Ketamine: A Novel Step in the Pathway from NMDA Receptor Blockade to Dopaminergic and Cognitive Disruptions Associated with the Prefrontal Cortex , 1997, The Journal of Neuroscience.
[31] F. Razoux,et al. Ketamine, at a Dose that Disrupts Motor Behavior and Latent Inhibition, Enhances Prefrontal Cortex Synaptic Efficacy and Glutamate Release in the Nucleus Accumbens , 2007, Neuropsychopharmacology.
[32] J. Deakin,et al. Asymmetrical reductions of hippocampal NMDAR1 glutamate receptor mRNA in the psychoses , 2001, Neuroreport.
[33] L. H. Finkel,et al. N-methyl-d-aspartic acid receptor antagonist–induced frequency oscillations in mice recreate pattern of electrophysiological deficits in schizophrenia , 2009, Neuroscience.
[34] Xianjin Zhou,et al. System-Wide Immunohistochemical Analysis of Protein Co-Localization , 2012, PloS one.
[35] D. McCormick,et al. Ionic Mechanisms Underlying Repetitive High-Frequency Burst Firing in Supragranular Cortical Neurons , 2000, The Journal of Neuroscience.
[36] Marc G Caron,et al. Mice with Reduced NMDA Receptor Expression Display Behaviors Related to Schizophrenia , 1999, Cell.
[37] Jessica A. Cardin,et al. A critical role for NMDA receptors in parvalbumin interneurons for gamma rhythm induction and behavior , 2011, Molecular Psychiatry.
[38] Hee-Sup Shin,et al. Mutant Mice and Neuroscience: Recommendations Concerning Genetic Background , 1997, Neuron.
[39] Jessica A. Cardin,et al. Driving fast-spiking cells induces gamma rhythm and controls sensory responses , 2009, Nature.
[40] J. Potash,et al. Novel loci for major depression identified by genome-wide association study of STAR*D and meta-analysis of three studies , 2009, Molecular Psychiatry.
[41] D J Porteous,et al. Schizophrenia and affective disorders--cosegregation with a translocation at chromosome 1q42 that directly disrupts brain-expressed genes: clinical and P300 findings in a family. , 2001, American journal of human genetics.
[42] Anil K Malhotra,et al. Ketamine-Induced Exacerbation of Psychotic Symptoms and Cognitive Impairment in Neuroleptic-Free Schizophrenics , 1997, Neuropsychopharmacology.
[43] Mark Jeremy Hunt,et al. Ketamine Dose-Dependently Induces High-Frequency Oscillations in the Nucleus Accumbens in Freely Moving Rats , 2006, Biological Psychiatry.
[44] Dheeraj Malhotra,et al. Reduced NMDAR1 expression in the Sp4 hypomorphic mouse may contribute to endophenotypes of human psychiatric disorders. , 2010, Human molecular genetics.
[45] M. Low,et al. Differential Contributions of Dopamine D1, D2, and D3 Receptors to MDMA-Induced Effects on Locomotor Behavior Patterns in Mice , 2006, Neuropsychopharmacology.
[46] Michael F. Green,et al. Analysis of 94 candidate genes and 12 endophenotypes for schizophrenia from the Consortium on the Genetics of Schizophrenia. , 2011, The American journal of psychiatry.
[47] Xiaoyu Peng,et al. Anti-NMDA-receptor encephalitis: case series and analysis of the effects of antibodies , 2008, The Lancet Neurology.
[48] Igor Jurisica,et al. CpG Island microarray probe sequences derived from a physical library are representative of CpG Islands annotated on the human genome , 2005, Nucleic acids research.
[49] M. Geyer,et al. Lamotrigine prevents ketamine but not amphetamine-induced deficits in prepulse inhibition in mice , 2003, Psychopharmacology.
[50] J. Haro,et al. The transcription factor SP4 is reduced in postmortem cerebellum of bipolar disorder subjects: control by depolarization and lithium , 2011, Bipolar disorders.